Fragility of bones than in men than women ...

Hip fractures in men account for one third of all hip fractures and have higher mortality than women. Burden on public health will increase with increasing number of older people in society increases. In addition, in the age incidence of hip fractures may increase in some but not all countries. Fractures of the spine may be a public health issue, as recent studies show that prevalence in the community is 20-30%, the same as in women. Forearm fractures probably should not be construed as a problem of public health. Peak bone mass is higher in men than women because men have more bone. Peak bone mineral density is the same. Number of spongy bone lost in the spine and crest of the ilium with aging is similar in men and women. Cork bone loss than men because endocortical resorption and periosteal less education or more. Loss of bone mass is accelerated in older men lasix furosemide side effects because endocortical resorption and increased porosity korkovoy increase the area intended for the resolution. Fragility of bones than in men than women because: (a) cross-sectional surface of the bone more, (b) loss of bone mass trabekulyarnoy less as a percentage of higher peak bone mass, (c) trabekulyarnoy bone loss occurs due to thinning and no perforation, and (d) periosteal appositional growth compensates for endocortical resorption, keeping bone bending strength. Reduced BMD in men with fractures may be due to reduction of peak bone mass and bone mass and loss. Loss of bone mass occurs by reducing bone formation. Do people with fractures increased bone fragility due to reduced periosteal growth attached to the process of aging is unknown. Reduction of the age level of testosterone, adrenal androgens, growth hormone and growth factor ynsulynopodobnoho 1 may help to reduce bone formation and bone loss. Men with fractures of the spine often hypogonadism or illnesses of several clinical signs that should be considered with a high index of suspicion (alcoholism, myeloma, malabsorption, primary hyperparathyroidism, hemochromatosis, pituitary Cushing's). Secondary hyperparathyroidism may contribute to bone loss by activating bone turnover and thus increase the number of bone remodeling units in violation of the formation of bones in each. There is no proven treatment for osteoporosis in men, because there were no trials using anti-fracture efficacy as an endpoint. Testosterone should be considered in men with hypogonadism and tested vitamin D deficiency should be corrected if present. Calcium supplements and bisphosphonates reasonable options given the lack of information. .